REHABILITATION OF MODERATE TO SEVERE TBI:
SENSORY-PERCEPTUAL AND BALANCE DISORDERS IN TBI: PERCEPTUAL
DEFICITS
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Agnosias - Deficits in stimulus or object recognition
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Forms:
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Apperceptive agnosia - impairment caused by disturbed integration
of otherwise normally perceived components of a stimulus
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Associative agnosia - defective recognition of normally perceived
and normally integrated percepts
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Types seen in TBI:
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Tactile agnosia - an inability to identify objects placed in the affected
hand (astereognosis), due to lesions of the parietal post-rolandic cortex;
common following TBI
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Auditory agnosia - a rare inability to differentiate between various
nonverbal sounds, probably due to bilateral temporal lobe lesions; rare and
usually occurs in conjunction with other auditory processing disorders
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Visual agnosia - a rare inability to recognize visual stimuli in otherwise
normal visual perception:
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Apperceptive visual agnosia may be correlated with dysfunction of the bilateral
visual association cortices
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Associative visual agnosia is a disconnection sydrome between the visual
and language systems in which the patient can recognize the object but is
unable to name it; may be due to occlusion of the left posterior cerebral
artery and subsequent infarction of the left occipital lobe and posterior
corpus callosum
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Prosopagnosia - an inability to recognize familiar faces and/or objects,
associated with nondominant parietal lobe involvement
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Frequently involves problems with color agnosia and alexia
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Enduring prosopagnosia is associated with bilateral lesions in the
inferomesial visual association cortices or their adjacent white matter,
due, in most cases, to embolization of the posterior cerebral artery circulation
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Finger agnosia may involve all 5 digits, but usually occurs in the
middle, index, and ring fingers, due, in most cases, to a lesion in the angular
gyrus of the dominant parietal lobe
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Neglect and Hemiinattention
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Neglect - an inability to report, respond, or orient to novel or
meaningful stimuli presented to the side opposite a brain lesion, due to
faulty mediation of both sensory and motor intention
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Unilateral neglect involves inattention to contralateral stimuli with
a lesser degree of ipsilateral inattention, and has been correlated with
lesions in the posterior parietal cortex, lateral prefrontal cortex, cingulate
gyrus, striatum, and thalamus
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Hemiinattention is the middle of the spectrum between extreme neglect
to extinction, only with double simultaneous stimulation
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Associated with neglect are:
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Anosognosia - the explicit verbal denial of illness or deficit
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Anosodiaphoria - verbal acknowledgement of the problem, but lack of
associated concern
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Alloesthesia - misplacement of the location of the stimulus to the
normal side, when the side contralateral to a lesion is stimulated
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Testing for neglect:
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Assess at least 3 sensory modalities, such as touch, vision, and audition
(unilateral hearing loss is almost aways due to peripheral dysfunction and
not hemiinattention)
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Present stimuli bilaterally, randomly, and simultaneously
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Recovery/Treatment:
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Neglect and hemiinattention tend to improve over time
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Rehabilitative approaches may be of some value, such as "anchoring" and
"scanning", vestibular stimulation, and bromocriptine
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Visuoperceptual Disorders
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Cortical blindness due to bilateral visual cortex damage;
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Anton's syndrome, a type of visual agnosia that involves the denial
of blindness, in which both the blindness and denial are transitory
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Balint's syndrome, an inability to perceive the entire visual field
which results in unpredictable perception and recognition of field components
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Marked by visual disorientation, optic ataxia (deficient visual reaching),
and ocular apraxia (visual scanning deficits)
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Associated symptoms include an inability to shift gaze at will toward new
stimuli and impaired target pointing under visual guidance
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Usually due to bilateral parietooccipital damage secondary to hypoperfusion
and infarctions of the posterior circulation
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Alexia, a disorder charcterized by recognition of disordered visual
patterns due to lesions that cause a disconnection between visual and language
centers
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Topographic disorientation, such as an inability to find one's way
back, is likely due to deficits in visuospatial memory and differs from
geographic disorientation, such as the inability to identify cities on a
map
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Achromatopsias are disorders of color perception
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Stereopsis is the inability to discern depth based on binocular vision
(as opposed to depth perception which is based on monocular vision)
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Visual hallucinations in patients with epileptic foci
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Based on information in Medical Rehabilitation
of Traumatic Brain Injury, L.J. Horn and N.D. Zasler, eds. St. Louis, MO,
Mosby, 1996, except for information where other papers are cited.
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